Dichlorphenamide

CHEMBL17 Phase 4 承認済み Small molecule
Half-Life
Bioavailability
Protein Binding
Molecular Weight
305.2 g/mol
LogP
0.3
Phase
4

A carbonic anhydrase inhibitor used to treat periodic paralysis, a condition causing episodes of muscle weakness. It works by affecting ion transport in muscles to prevent paralysis attacks.

分子量

305.2000 g/mol

LogP

0.30

TPSA

137.00 Ų

リピンスキーの五則

適合

作用機序

Selectively blocks angiotensin II type 1 (AT1) receptors, preventing the vasoconstrictive and aldosterone-secreting effects of angiotensin II. This results in vasodilation, reduced sodium retention, and decreased blood pressure.

Pharmacokinetics (PK)

Pharmacodynamics (PD)

機序

Selectively blocks angiotensin II type 1 (AT1) receptors, preventing the vasoconstrictive and aldosterone-secreting effects of angiotensin II. This results in vasodilation, reduced sodium retention, and decreased blood pressure.

2D構造

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SMILES

NS(=O)(=O)c1cc(Cl)c(Cl)c(S(N)(=O)=O)c1

InChI

InChI=1S/C6H6Cl2N2O4S2/c7-4-1-3(15(9,11)12)2-5(6(4)8)16(10,13)14/h1-2H,(H2,9,11,12)(H2,10,13,14)

Molecular Formula

C6H6Cl2N2O4S2

HBD / HBA

2 / 6

回転可能結合数

2

重原子数

16

No targets recorded

Target interaction data is not yet available for this drug.

No interactions recorded

Drug interaction data is not yet available for this compound.

No side effects recorded

Side effect data is not yet available for this drug.

よくある質問

A carbonic anhydrase inhibitor used to treat periodic paralysis, a condition causing episodes of muscle weakness. It works by affecting ion transport in muscles to prevent paralysis attacks.

Selectively blocks angiotensin II type 1 (AT1) receptors, preventing the vasoconstrictive and aldosterone-secreting effects of angiotensin II. This results in vasodilation, reduced sodium retention, and decreased blood pressure.

Yes, Dichlorphenamide is an approved drug. It has reached clinical phase 4. It is classified as a Small molecule.

{# References & Data Sources section for drug detail pages. Renders standard pharmacological database links plus the drug's data_sources field. #}

References & Data Sources

  • ChEMBL — European Bioinformatics Institute (EBI). CHEMBL17. Open-access bioactivity database.
  • PubChem — National Center for Biotechnology Information (NCBI). CID 3038. Chemical information database.

Data aggregated from publicly available pharmacological databases. Last updated 2026-03-04.

医学的免責事項

This content is for educational and informational purposes only. It is not a substitute for professional medical advice, diagnosis, or treatment. Always consult a qualified healthcare provider before making medication decisions.

Data sources: ChEMBL, PubChem, DailyMed.