Anileridine Hydrochloride
The hydrochloride salt of anileridine, sharing the same opioid pain-relieving mechanism as the parent compound by activating mu-opioid receptors. It has largely been replaced by other opioids in modern practice.
Peso Molecular
388,9000 g/mol
TPSA
55,60 Ų
Mecanismo de Ação
Binds to mu (μ), kappa (κ), and/or delta (δ) opioid receptors in the central and peripheral nervous system, mimicking endogenous endorphins. Activation of these G-protein-coupled receptors inhibits pain signal transmission and modulates the emotional response to pain.
Pharmacokinetics (PK)
Pharmacodynamics (PD)
Binds to mu (μ), kappa (κ), and/or delta (δ) opioid receptors in the central and peripheral nervous system, mimicking endogenous endorphins. Activation of these G-protein-coupled receptors inhibits pain signal transmission …
Estrutura 2D
Cite this structure
Embed this structure
SMILES
CCOC(=O)C1(c2ccccc2)CCN(CCc2ccc(N)cc2)CC1.Cl
InChI
InChI=1S/C22H28N2O2.ClH/c1-2-26-21(25)22(19-6-4-3-5-7-19)13-16-24(17-14-22)15-12-18-8-10-20(23)11-9-18;/h3-11H,2,12-17,23H2,1H3;1H
Molecular Formula
C22H29ClN2O2
HBD / HBA
2 / 4
Ligações Rotacionáveis
7
Átomos Pesados
27
No targets recorded
Target interaction data is not yet available for this drug.
No interactions recorded
Drug interaction data is not yet available for this compound.
No side effects recorded
Side effect data is not yet available for this drug.
Perguntas frequentes
The hydrochloride salt of anileridine, sharing the same opioid pain-relieving mechanism as the parent compound by activating mu-opioid receptors. It has largely been replaced by other opioids in modern practice.
Binds to mu (μ), kappa (κ), and/or delta (δ) opioid receptors in the central and peripheral nervous system, mimicking endogenous endorphins. Activation of these G-protein-coupled receptors inhibits pain signal transmission and modulates the emotional response to pain.
Yes, Anileridine Hydrochloride is an approved drug. It has reached clinical phase 4. It is classified as a Small molecule.
References & Data Sources
- ChEMBL — European Bioinformatics Institute (EBI). CHEMBL1201047. Open-access bioactivity database.
- PubChem — National Center for Biotechnology Information (NCBI). CID 197194. Chemical information database.
Data aggregated from publicly available pharmacological databases. Last updated 2026-03-04.
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