Carbamoylcholine
Another name for carbachol, a cholinomimetic agent that activates both muscarinic and nicotinic acetylcholine receptors. It is used as an ophthalmic agent to constrict the pupil and lower intraocular pressure. It is resistant to cholinesterase-mediated breakdown, resulting in a prolonged duration of action.
Moleküler Ağırlık
147,2000 g/mol
LogP
-0,40
TPSA
52,30 Ų
Lipinski RO5
Geçer
Etki Mekanizması
Selectively blocks angiotensin II type 1 (AT1) receptors, preventing the vasoconstrictive and aldosterone-secreting effects of angiotensin II. This results in vasodilation, reduced sodium retention, and decreased blood pressure.
Pharmacokinetics (PK)
Pharmacodynamics (PD)
Selectively blocks angiotensin II type 1 (AT1) receptors, preventing the vasoconstrictive and aldosterone-secreting effects of angiotensin II. This results in vasodilation, reduced sodium retention, and decreased blood pressure.
2D Yapı
Cite this structure
Embed this structure
SMILES
C[N+](C)(C)CCOC(N)=O
InChI
InChI=1S/C6H14N2O2/c1-8(2,3)4-5-10-6(7)9/h4-5H2,1-3H3,(H-,7,9)/p+1
Molecular Formula
C6H15N2O2+
HBD / HBA
1 / 2
Döndürülebilir Bağlar
4
Ağır Atomlar
10
No targets recorded
Target interaction data is not yet available for this drug.
No interactions recorded
Drug interaction data is not yet available for this compound.
No side effects recorded
Side effect data is not yet available for this drug.
Sıkça Sorulan Sorular
Another name for carbachol, a cholinomimetic agent that activates both muscarinic and nicotinic acetylcholine receptors. It is used as an ophthalmic agent to constrict the pupil and lower intraocular pressure. It is resistant to cholinesterase-mediated breakdown, resulting in a prolonged duration of action.
Selectively blocks angiotensin II type 1 (AT1) receptors, preventing the vasoconstrictive and aldosterone-secreting effects of angiotensin II. This results in vasodilation, reduced sodium retention, and decreased blood pressure.
Yes, Carbamoylcholine is an approved drug. It has reached clinical phase 4. It is classified as a Small molecule.
References & Data Sources
- ChEMBL — European Bioinformatics Institute (EBI). CHEMBL965. Open-access bioactivity database.
- PubChem — National Center for Biotechnology Information (NCBI). CID 2551. Chemical information database.
Data aggregated from publicly available pharmacological databases. Last updated 2026-03-04.
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